Hello everybody,
I have COPD, not CF, but have been told I could post here; and I've gotten help from you folks in the past, and appreciate it.
I've had a p.a. colony since 2003, and after a culture last Sept grew it I was put on TOBI for 28 days, and then switched to Colistin. There was no active infection. However I have now been on Colistin since Oct 10 without a break. During this time, and especially around Xmas inflammation affected my breathing in a major way, and that's why the doc ordered th third month of Colistin, which along with a lot of prednisone seems to be bringing me around.
My question is: is/was the p.a. likely to have been the reason for the increase in inflammation, without other signs of infection like purulent sputum or is it just a roll of the dice? Also, shoudln't (ideally) I be rotating the Colistin on/off a month at a time rather than just staying on it steadily?
Thanks for any comment, and keep up the good work!
I have COPD, not CF, but have been told I could post here; and I've gotten help from you folks in the past, and appreciate it.
I've had a p.a. colony since 2003, and after a culture last Sept grew it I was put on TOBI for 28 days, and then switched to Colistin. There was no active infection. However I have now been on Colistin since Oct 10 without a break. During this time, and especially around Xmas inflammation affected my breathing in a major way, and that's why the doc ordered th third month of Colistin, which along with a lot of prednisone seems to be bringing me around.
My question is: is/was the p.a. likely to have been the reason for the increase in inflammation, without other signs of infection like purulent sputum or is it just a roll of the dice? Also, shoudln't (ideally) I be rotating the Colistin on/off a month at a time rather than just staying on it steadily?
Thanks for any comment, and keep up the good work!