FEV1, O2 Sat with and without exercise

[Wendy]

FEV1: 62% pre-pregnancy (currently 55% at 38 weeks pregnant)
O2 Sat resting: 98%
02 Sat while exercising: 92%-96% (only ever checked in hospital for tune-ups)
HR resting: 68-75

Years ago I ran alot. Not so much lateley. I had just gotten back into a steady workout routine in September and became pregnant that month. I continued to exercise up until my 30th week of pregnancy and then stopped because it was taking too much out of me the next day. I think some people's bodies just tolerate exercise better than others. Barring pregnancy, I have a pretty low heartrate and tolerate exercise well with little oxygen de-satting (even when I'm sick). My heartrate during exercise generally doesn't go over 145 (if I remember correctly), even at a good run.

 

[Wendy]

FEV1: 62% pre-pregnancy (currently 55% at 38 weeks pregnant)
O2 Sat resting: 98%
02 Sat while exercising: 92%-96% (only ever checked in hospital for tune-ups)
HR resting: 68-75

Years ago I ran alot. Not so much lateley. I had just gotten back into a steady workout routine in September and became pregnant that month. I continued to exercise up until my 30th week of pregnancy and then stopped because it was taking too much out of me the next day. I think some people's bodies just tolerate exercise better than others. Barring pregnancy, I have a pretty low heartrate and tolerate exercise well with little oxygen de-satting (even when I'm sick). My heartrate during exercise generally doesn't go over 145 (if I remember correctly), even at a good run.

 

[Wendy]

FEV1: 62% pre-pregnancy (currently 55% at 38 weeks pregnant)
O2 Sat resting: 98%
02 Sat while exercising: 92%-96% (only ever checked in hospital for tune-ups)
HR resting: 68-75

Years ago I ran alot. Not so much lateley. I had just gotten back into a steady workout routine in September and became pregnant that month. I continued to exercise up until my 30th week of pregnancy and then stopped because it was taking too much out of me the next day. I think some people's bodies just tolerate exercise better than others. Barring pregnancy, I have a pretty low heartrate and tolerate exercise well with little oxygen de-satting (even when I'm sick). My heartrate during exercise generally doesn't go over 145 (if I remember correctly), even at a good run.

 

[Wendy]

FEV1: 62% pre-pregnancy (currently 55% at 38 weeks pregnant)
O2 Sat resting: 98%
02 Sat while exercising: 92%-96% (only ever checked in hospital for tune-ups)
HR resting: 68-75

Years ago I ran alot. Not so much lateley. I had just gotten back into a steady workout routine in September and became pregnant that month. I continued to exercise up until my 30th week of pregnancy and then stopped because it was taking too much out of me the next day. I think some people's bodies just tolerate exercise better than others. Barring pregnancy, I have a pretty low heartrate and tolerate exercise well with little oxygen de-satting (even when I'm sick). My heartrate during exercise generally doesn't go over 145 (if I remember correctly), even at a good run.

 

[Wendy]

FEV1: 62% pre-pregnancy (currently 55% at 38 weeks pregnant)
O2 Sat resting: 98%
02 Sat while exercising: 92%-96% (only ever checked in hospital for tune-ups)
HR resting: 68-75

Years ago I ran alot. Not so much lateley. I had just gotten back into a steady workout routine in September and became pregnant that month. I continued to exercise up until my 30th week of pregnancy and then stopped because it was taking too much out of me the next day. I think some people's bodies just tolerate exercise better than others. Barring pregnancy, I have a pretty low heartrate and tolerate exercise well with little oxygen de-satting (even when I'm sick). My heartrate during exercise generally doesn't go over 145 (if I remember correctly), even at a good run.

 

[Wendy]

FEV1: 62% pre-pregnancy (currently 55% at 38 weeks pregnant)
O2 Sat resting: 98%
02 Sat while exercising: 92%-96% (only ever checked in hospital for tune-ups)
HR resting: 68-75

Years ago I ran alot. Not so much lateley. I had just gotten back into a steady workout routine in September and became pregnant that month. I continued to exercise up until my 30th week of pregnancy and then stopped because it was taking too much out of me the next day. I think some people's bodies just tolerate exercise better than others. Barring pregnancy, I have a pretty low heartrate and tolerate exercise well with little oxygen de-satting (even when I'm sick). My heartrate during exercise generally doesn't go over 145 (if I remember correctly), even at a good run.

 

[ladybug]

This would be a great correlational or longitudinal study by CFF since exercise is such an integral part of our care plan. I would have thought there would be at least ONE published article with regard to CF and exercise O2 sats or correlational studies that would link FEV1 with O2 sats or something clever like that. Its odd to me that CFF doesn't make more use of its database and extend it to things like this so docs as well as patients could have this invaluable information at their fingertips.

I certainly think these sort of threads that ask for info. on FEV1 and exercise or O2 and excercise, etc. would be GREAT things the CFF could keep track of, or even just a local clinic plotting it (if they actually had us tested that is!) and maybe published or something. I don't know. I just think its odd that WE have the inquisitive minds to ask such questions and seek answers, but no one else in the scientific community has compiled such data. Hmmm....

Just a thought with all the talk about exercise and what's "normal". <img src="i/expressions/face-icon-small-smile.gif" border="0">

 

[ladybug]

This would be a great correlational or longitudinal study by CFF since exercise is such an integral part of our care plan. I would have thought there would be at least ONE published article with regard to CF and exercise O2 sats or correlational studies that would link FEV1 with O2 sats or something clever like that. Its odd to me that CFF doesn't make more use of its database and extend it to things like this so docs as well as patients could have this invaluable information at their fingertips.

I certainly think these sort of threads that ask for info. on FEV1 and exercise or O2 and excercise, etc. would be GREAT things the CFF could keep track of, or even just a local clinic plotting it (if they actually had us tested that is!) and maybe published or something. I don't know. I just think its odd that WE have the inquisitive minds to ask such questions and seek answers, but no one else in the scientific community has compiled such data. Hmmm....

Just a thought with all the talk about exercise and what's "normal". <img src="i/expressions/face-icon-small-smile.gif" border="0">

 

[ladybug]

This would be a great correlational or longitudinal study by CFF since exercise is such an integral part of our care plan. I would have thought there would be at least ONE published article with regard to CF and exercise O2 sats or correlational studies that would link FEV1 with O2 sats or something clever like that. Its odd to me that CFF doesn't make more use of its database and extend it to things like this so docs as well as patients could have this invaluable information at their fingertips.

I certainly think these sort of threads that ask for info. on FEV1 and exercise or O2 and excercise, etc. would be GREAT things the CFF could keep track of, or even just a local clinic plotting it (if they actually had us tested that is!) and maybe published or something. I don't know. I just think its odd that WE have the inquisitive minds to ask such questions and seek answers, but no one else in the scientific community has compiled such data. Hmmm....

Just a thought with all the talk about exercise and what's "normal". <img src="i/expressions/face-icon-small-smile.gif" border="0">

 

[ladybug]

This would be a great correlational or longitudinal study by CFF since exercise is such an integral part of our care plan. I would have thought there would be at least ONE published article with regard to CF and exercise O2 sats or correlational studies that would link FEV1 with O2 sats or something clever like that. Its odd to me that CFF doesn't make more use of its database and extend it to things like this so docs as well as patients could have this invaluable information at their fingertips.

I certainly think these sort of threads that ask for info. on FEV1 and exercise or O2 and excercise, etc. would be GREAT things the CFF could keep track of, or even just a local clinic plotting it (if they actually had us tested that is!) and maybe published or something. I don't know. I just think its odd that WE have the inquisitive minds to ask such questions and seek answers, but no one else in the scientific community has compiled such data. Hmmm....

Just a thought with all the talk about exercise and what's "normal". <img src="i/expressions/face-icon-small-smile.gif" border="0">

 

[ladybug]

This would be a great correlational or longitudinal study by CFF since exercise is such an integral part of our care plan. I would have thought there would be at least ONE published article with regard to CF and exercise O2 sats or correlational studies that would link FEV1 with O2 sats or something clever like that. Its odd to me that CFF doesn't make more use of its database and extend it to things like this so docs as well as patients could have this invaluable information at their fingertips.

I certainly think these sort of threads that ask for info. on FEV1 and exercise or O2 and excercise, etc. would be GREAT things the CFF could keep track of, or even just a local clinic plotting it (if they actually had us tested that is!) and maybe published or something. I don't know. I just think its odd that WE have the inquisitive minds to ask such questions and seek answers, but no one else in the scientific community has compiled such data. Hmmm....

Just a thought with all the talk about exercise and what's "normal". <img src="i/expressions/face-icon-small-smile.gif" border="0">

 

[ladybug]

This would be a great correlational or longitudinal study by CFF since exercise is such an integral part of our care plan. I would have thought there would be at least ONE published article with regard to CF and exercise O2 sats or correlational studies that would link FEV1 with O2 sats or something clever like that. Its odd to me that CFF doesn't make more use of its database and extend it to things like this so docs as well as patients could have this invaluable information at their fingertips.

I certainly think these sort of threads that ask for info. on FEV1 and exercise or O2 and excercise, etc. would be GREAT things the CFF could keep track of, or even just a local clinic plotting it (if they actually had us tested that is!) and maybe published or something. I don't know. I just think its odd that WE have the inquisitive minds to ask such questions and seek answers, but no one else in the scientific community has compiled such data. Hmmm....

Just a thought with all the talk about exercise and what's "normal". <img src="i/expressions/face-icon-small-smile.gif" border="0">

 

[NoExcuses]

i have a feeling there is no data because there isn't much of a correlation.

different people sat at different levels - at different FEV1 levels.

so that information would be useless if it doesn't help us predict or decide when O2 is needed.

 

[NoExcuses]

i have a feeling there is no data because there isn't much of a correlation.

different people sat at different levels - at different FEV1 levels.

so that information would be useless if it doesn't help us predict or decide when O2 is needed.

 

[NoExcuses]

i have a feeling there is no data because there isn't much of a correlation.

different people sat at different levels - at different FEV1 levels.

so that information would be useless if it doesn't help us predict or decide when O2 is needed.

 

[NoExcuses]

i have a feeling there is no data because there isn't much of a correlation.

different people sat at different levels - at different FEV1 levels.

so that information would be useless if it doesn't help us predict or decide when O2 is needed.

 

[NoExcuses]

i have a feeling there is no data because there isn't much of a correlation.

different people sat at different levels - at different FEV1 levels.

so that information would be useless if it doesn't help us predict or decide when O2 is needed.

 

[NoExcuses]

i have a feeling there is no data because there isn't much of a correlation.

different people sat at different levels - at different FEV1 levels.

so that information would be useless if it doesn't help us predict or decide when O2 is needed.

 

[LisaV]

hyperinflation (hy·per·in·fla·tion) (hi"p?r-in-fla´sh?n) excessive inflation or expansion, as of the lungs; called also overinflation. (One symptom of this is the ever expanding ribcage) From <a target=_blank class=ftalternatingbarlinklarge href="http://www.medhelp.org/forums/RespiratoryDisorders/messages/919.html">http://www.medhelp.org/forums/...ders/messages/919.html</a> "Hyperinflated lungs mean air is trapped in the small airways and the lungs appear larger than usual, on the chest X-ray. Hyperinflation is not specific for COPD. It can be seen with asthma and other lung diseases. If it occurs in only one lung, obstruction of the bronchus to that lung should be considered"
See also <a target=_blank class=ftalternatingbarlinklarge href="http://www.medscape.com/viewarticle/516202">http://www.medscape.com/viewarticle/516202</a> which discusses this and the difference between resting hyperinflation and dynamic (during exercise) hyperinflation.
I have never seen the lung reduction surgery recommended for someone with cystic fibrosis.

Article on Pulmonary Hypertension and Cor Pulmonale in COPD <a target=_blank class=ftalternatingbarlinklarge href="http://www.medscape.com/viewarticle/458660_6">http://www.medscape.com/viewarticle/458660_6</a> . Another on the Treatment of Pulmonary Hypertension Related to Disorders of hypoxia at <a target=_blank class=ftalternatingbarlinklarge href="http://www.phassociation.org/Medical/Advances_in_PH/Summer_2005/hypoxia.asp">http://www.phassociation.org/M...ummer_2005/hypoxia.asp</a> . From <a target=_blank class=ftalternatingbarlinklarge href="http://pats.atsjournals.org/cgi/content/full/2/1/12">http://pats.atsjournals.org/cgi/content/full/2/1/12</a> "It is first important to know how many patients with severe COPD have PH and to know to what degree the PH affects them. Despite many uncertainties, studies indicate that 35% of patients with severe COPD have pulmonary artery pressures (Ppa) of more than 20 mm Hg at rest (38). Of those patients without PH at rest, a further 52% will develop PH during exercise (39). Furthermore, other reports propose that 91% of patients with severe COPD have PH (56). Exercise-induced rises in Ppa predict those patients who will eventually develop PH at rest (39), and are probably associated with exercise-induced hypoxia (57). "

I agree with Amy that it is very individual. I would also point out that have cystic fibrosis does not immunize one from asthma and exercise-induced asthma hypoxia is not at all uncommon.

Also I'm sort of surprised by the lack of information you all have been given about the benefits of using O2 early to avoid pulmonary hypertension and Cor Pulmonale. I can't help but wonder if this isn't because folks with cystic fibrosis used to die so early that it wasn't worth putting them on oxygen because years ago it was so freaky to see a little kid on those big tanks (while now we're talking about little tanks on the waists of grownups).

 

[LisaV]

hyperinflation (hy·per·in·fla·tion) (hi"p?r-in-fla´sh?n) excessive inflation or expansion, as of the lungs; called also overinflation. (One symptom of this is the ever expanding ribcage) From <a target=_blank class=ftalternatingbarlinklarge href="http://www.medhelp.org/forums/RespiratoryDisorders/messages/919.html">http://www.medhelp.org/forums/...ders/messages/919.html</a> "Hyperinflated lungs mean air is trapped in the small airways and the lungs appear larger than usual, on the chest X-ray. Hyperinflation is not specific for COPD. It can be seen with asthma and other lung diseases. If it occurs in only one lung, obstruction of the bronchus to that lung should be considered"
See also <a target=_blank class=ftalternatingbarlinklarge href="http://www.medscape.com/viewarticle/516202">http://www.medscape.com/viewarticle/516202</a> which discusses this and the difference between resting hyperinflation and dynamic (during exercise) hyperinflation.
I have never seen the lung reduction surgery recommended for someone with cystic fibrosis.

Article on Pulmonary Hypertension and Cor Pulmonale in COPD <a target=_blank class=ftalternatingbarlinklarge href="http://www.medscape.com/viewarticle/458660_6">http://www.medscape.com/viewarticle/458660_6</a> . Another on the Treatment of Pulmonary Hypertension Related to Disorders of hypoxia at <a target=_blank class=ftalternatingbarlinklarge href="http://www.phassociation.org/Medical/Advances_in_PH/Summer_2005/hypoxia.asp">http://www.phassociation.org/M...ummer_2005/hypoxia.asp</a> . From <a target=_blank class=ftalternatingbarlinklarge href="http://pats.atsjournals.org/cgi/content/full/2/1/12">http://pats.atsjournals.org/cgi/content/full/2/1/12</a> "It is first important to know how many patients with severe COPD have PH and to know to what degree the PH affects them. Despite many uncertainties, studies indicate that 35% of patients with severe COPD have pulmonary artery pressures (Ppa) of more than 20 mm Hg at rest (38). Of those patients without PH at rest, a further 52% will develop PH during exercise (39). Furthermore, other reports propose that 91% of patients with severe COPD have PH (56). Exercise-induced rises in Ppa predict those patients who will eventually develop PH at rest (39), and are probably associated with exercise-induced hypoxia (57). "

I agree with Amy that it is very individual. I would also point out that have cystic fibrosis does not immunize one from asthma and exercise-induced asthma hypoxia is not at all uncommon.

Also I'm sort of surprised by the lack of information you all have been given about the benefits of using O2 early to avoid pulmonary hypertension and Cor Pulmonale. I can't help but wonder if this isn't because folks with cystic fibrosis used to die so early that it wasn't worth putting them on oxygen because years ago it was so freaky to see a little kid on those big tanks (while now we're talking about little tanks on the waists of grownups).